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brand new study touts relationship between fibromyalgia and insulin resistance

STRUGGLING WITH FIBROMYALGIA?
SOLVING YOUR BLOOD SUGAR ISSUES COULD PROVE HELPFUL

Blood Sugar Fibromyalgia

I’ve shown you time and time again that blood sugar dysregulation is the primary source of inflammation behind most health problems (HERE).  I’ve also shown you that VIA THESE INFLAMMATORY PATHWAYS, most health problems can actually be tied back to blood sugar dysregulation via studies / peer review (start looking at links on this page for proof).  This relationship alone should help you better understand the chasm seen between medical research and medical practice (HERE).

Earlier this week the medical journal PLoS One published a study by an eight-member team (three from University of Texas, LSU, the NIH, St. Michael’s Pain & Spine Clinics, College of Osteopathic Medicine of the Pacific, and Alaska’s Pain and Headache Center of Eagle River) titled Is insulin Resistance the Cause of Fibromyalgia? A Preliminary Report.  Could these experts be right?  Could BLOOD SUGAR DYSREGULATION be a causal factor for developing FIBROMYALGIA?

The authors began by discussing the cost of fibromyalgia (over 100 billion dollars annually in the United States alone), incidence (nearly 3% of the worldwide population — undoubtedly underestimated in America — with three times more women than men affected). What sort of symptoms are we talking about here?

Fibromyalgia symptoms range from MUSCULOSKELETAL PAIN, CHRONIC HEADACHES, SPINAL DISORDERS and CHRONIC NECK PAIN, CHRONIC FATIGUE, SLEEPING DISORDERS, mood disorders (ANXIETY, DEPRESSION, etc), problems with cognition, commonly referred to as brain fog or fibro fog {HERE}, and SMALL FIBER NEUROPATHY.

This unholy combination forms the basis of a recipe that makes fibromyalgia a “central sensitivity pain disorder characterized by abnormal processing of nociceptive stimuli” (CLICK HERE to understand the seriousness of centralized pain).   The biggest problem is that the medical community at large has little to offer these people.

“Many hypotheses have been advanced to explain the extensive array of symptoms including inherited abnormalities, dysfunction of neurotransmitter pathways such as substance P, immune dysregulation and several others. Unfortunately, none of these propositions has led to practical advances beyond symptomatic treatment. In fact, recent reviews of FM published in 2016 and 2017, have concluded that there have been no substantive advances in our understanding of this disease.”

While these theories have merit (you could have thrown in ADRENAL FATIGUE AS WELL), none have helped create mainstream medical solutions, although adrenal fatigue, as the link shows, is intimately related to consumption of sugar and highly processed carbohydrates.  Allow me to show you the highly intriguing theory put forth by these authors; neurologists, anesthesiologists, pharmacologists, etc.  

It’s a well known fact that INSULIN RESISTANCE causes a diminished perfusion of oxygen to certain parts of the brain.  Previous studies have also shown that similar microvascular damage is seen in fibro sufferer’s brains.

The authors combed through medical records of people diagnosed with fibromyalgia and found that, “patients with FM belong to a distinct population that can be segregated from a control group by their HbA1c values, a biomarker for impaired glucose metabolism, characterized by insulin resistance.”  If the patients had a hemoglobin A1c value over 5.7% they were, along with their regular regimen of drugs, given the ever-popular diabetes drug, metformin. 

The bottom line is that these authors not only found a strong correlation between higher HbA1c readings and worse fibromyalgia symptoms, they found they could substantially reduce patients FM symptoms by adding metformin to whatever COCKTAIL of fibro drugs they were already taking.  Half of the subjects reported a pain reduction of at least 50%.   Their conclusions?

“Insulin resistance is being increasingly associated with a broad number of neurological disorders……  Based on our data, we would like to propose that insulin resistance is pathogenetically linked to fibromyalgia.  It would be unlikely for a placebo type of effect alone to result in the impressive degree of long-lasting pain improvement experienced by the patients who received the drug combination (metformin plus standard treatment). Remarkably, ‘non-responders’ were conspicuously absent in our patient’s sample.”

Let’s discuss. 

Firstly, knowing what we know about diabetes drugs, do you really want to be taking them, espeically long-term, if there is a better way (HERE)?  Secondly, the list of links I provided earlier pertaining to FM symptoms clearly shows that most — PERIPHERAL NEUROPATHY INCLUDED — are heavily linked to sugar intake.  

And thirdly, I just wrote an extremely cool post about the fact that the “vascular damage” done by “hypoperfusion of oxygen” (these author’s words for describing what’s taking place in a sugar-saturated brain) is REVERSIBLE WITH EWOT.  That is reversible as in reversible.

We can’t be surprised by these conclusions considering that sugar has long been known to cause problems (especially thickening) of connective tissues including fascia — HERE, HERE, HERE, HERE or HERE).  And rather than providing FM sufferers an excuse to take more drugs, this study should motivate them to address their issue(s) without drugs. 

As you can see from MY HEALTH & INFLAMMATION PROTOCOL; arguably the best means of addressing all health-related involves maintaining a steady state of blood sugar.  It’s why PALEO, KETO, or a combination of both (HERE), can be nothing short of miraculous for chronically ill people, fibro sufferers included. 

Because we all know plenty of people struggling with chronic conditions, including diabetes, obesity (diabesity) or even HYPOGLYCEMIA, be sure to spread the wealth by getting this info in front of their faces.  A great way to reach the people you love and value most?  FACEBOOK.

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One Response

  1. I’d be interested to know what is going on in FM sufferers who are very prone to weight gain, which I assume to be consistent with insulin resistance – versus FM sufferers who are scrawny and cannot gain weight. I have always advocated a Keto diet to other FM sufferers, but some of them are scrawny rather than bulky like I am. What might a Keto diet to do them? Often they have temperamental digestive systems and can’t handle certain foods that to me, are staples of Keto diets.

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